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NDT Plus Advance Access originally published online on September 15, 2008
NDT Plus 2008 1(6):420-422; doi:10.1093/ndtplus/sfn147
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© The Author [2008]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org

Complete remission of IgA nephropathy after bone marrow transplantation for acute myeloid leukaemia

Eun-Kyung Park, Jin-Seok Jeon, Hyun-Jin Noh, Jong-Ho Won and Hee-Sook Park

Department of Internal Medicine, University of Soon Chun Hyang School of Medicine, Seoul, Korea

Correspondence: Correspondence and offprint requests to: Jong-Ho Won, Department of Internal Medicine, University of Soon Chun Hyang School of Medicine, 657 Hannam Dong, 140-743 Seoul, Korea. Tel: +82-2-709-9203; Fax: +82-2-792-5812; E-mail: jhwon{at}hosp.sch.ac.kr


  Abstract

IgA nephropathy is the most common primary glomerulonephritis, but the pathogenesis of IgA nephropathy is still unclear. A 32-year-old woman was found to have IgA nephropathy and acute myeloid leukaemia. She was treated with allogenic bone marrow transplantation (BMT). After BMT, immunoflourescent staining of IgA and proteinuria disappeared. These findings suggest bone marrow cells are involved in the pathogenesis of IgA nephropathy. We herein report a case of complete remission of IgA nephropathy after BMT for acute myeloid leukaemia.

Key Words: bone marrow transplantation • IgA nephropathy

Received for publication August 11, 2008. Accepted for publication August 13, 2008.


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