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NDT Plus Advance Access originally published online on November 21, 2008
NDT Plus 2009 2(1):72-75; doi:10.1093/ndtplus/sfn171
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© The Author [2008]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org

Fluindione-induced immuno-allergic interstitial nephritis

Rania Kheder-Elfekih1, Caroline Poitou1, Isabelle Brocheriou2, Helene Depreneuf3 and Hassane Izzedine1

1 Department of Nephrology
2 Department of Pathology, Pitie-Salpetiere Hospital
3 Department of Nephrology, AURA, Paris, France

Correspondence: Correspondence and offprint requests to: Hassane Izzedine, Department of Nephrology, La Pitié-Salpêtrière Hospital, 47-80 Boulevard de l’Hôpital, Assistance Publique-Hôpitaux de Paris, Pierre et Marie Curie University, 75013 Paris, France. Tel: +33142177225; Fax: +33142177232; E-mail: hassan.izzedine@psl.aphp.fr

Key Words: acute interstitial nephritis • corticosteroids • fluindione

Received for publication July 18, 2008. Accepted for publication October 21, 2008.

The first 10% of the full text of this article appears below.

Drug-induced acute interstitial nephritis (AIN) is an established cause of acute kidney injury (AKI). Antibiotics and non-steroidal anti-inflammatory drugs (NSAIDs) are the most frequent offending drugs [1]. Only a few vitamin K antagonist-induced AIN cases have been reported. Some publications describe AIN associated with fluindione (Previscan®), an anticoagulant of the antivitamin K family, derived from indanedione, exclusively marketed in France. We present an additional case of AIN secondary to fluindione and review the . . . [Full Text of this Article]


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