Skip Navigation

NDT Plus 2008 1(Supplement 1):i1; doi:10.1093/ndtplus/sfm036
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Google Scholar
Right arrow Articles by Carrera, F.
Right arrow Search for Related Content
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author [2007].
The online version of this article has been published under an open access model. Users are entitled to use, reproduce, disseminate, or display the open access version of this article for non-commercial purposes provided that: the original authorship is properly and fully attributed; the Journal and Oxford University Press are attributed as the original place of publication with the correct citation details given; if an article is subsequently reproduced or disseminated not in its entirety but only in part or as a derivative work this must be clearly indicated. For commercial re-use, please contact journals.permissions@oxfordjournals.org

Foreword

Fernando Carrera

Eurodial, Euromedic, Dialysis Unit, Leiria, Portugal

Correspondence: E-mail: fcarrera@mail.telepac.pt

Received for publication July 17, 2007. Accepted for publication September 10, 2007.

The first 10% of the full text of this article appears below.

Secondary hyperparathyroidism (SHPT) develops in chronic kidney disease (CKD) as a consequence of disturbances in mineral metabolism and vitamin D synthesis [1]. Alterations in signalling via the parathyroid gland calcium-sensing receptor (CaR) are known to play a central role in the development of SHPT [2,3], making . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?