Skip Navigation


NDT Plus Advance Access originally published online on October 13, 2008
NDT Plus 2008 1(6):440-441; doi:10.1093/ndtplus/sfn158
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow All Versions of this Article:
1/6/440    most recent
sfn158v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Connor, A.
Right arrow Articles by Taylor, J. E.
Right arrow Search for Related Content
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author [2008]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org

Renal impairment resulting from hypothyroidism

Andrew Connor and Joanne E. Taylor

Department of Renal Medicine, Dorset County Hospital, Dorchester, DT1 2JY, UK

Correspondence: Correspondence and offprint requests to: Andrew Connor, 3 Hope Terrace, Martinstown, Dorset, DT2 9JN, UK. Tel: +44-1305-889083; Fax: +44-1432265944; E-mail: andrewconnor1974@hotmail.co.uk

Key Words: hypothyroidism • renal impairment

Received for publication September 14, 2008. Accepted for publication September 17, 2008.

The first 10% of the full text of this article appears below.


    Introduction
 
We describe two cases of reversible renal impairment secondary to hypothyroidism. We suggest that patients with renal impairment of unknown cause have thyroid function tests undertaken as part of routine investigation.


    Case one
 
A 37-year-old woman presented with recent constitutional upset and a year-long history of swelling of her face and hands. Her past medical history included pre-eclampsia. Her father had suffered from focal segmental glomerulosclerosis and her brother underwent renal transplantation for reflux nephropathy. Examination revealed peri-orbital oedema and hypertension (140/100 mmHg). Her pulse rate was 58 beats/min.

Investigations included haemoglobin 11.6 g/l, white cell count 6.2 x 109/l, sodium 137 mmol/l, potassium 3.6 mmol/l, creatinine 148 µmol/l, . . . [Full Text of this Article]


    Case two
 

    Discussion
 

    Teaching points
 

Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?